Search results for "Nasal polyps"

showing 10 items of 38 documents

Fungal DNA is present in tissue specimens of patients with chronic rhinosinusitis.

2004

Background It has been postulated that fungal organisms might represent the immunologic target initiating and maintaining the disease process in patients with chronic rhinosinusitis (CRS). The presence of fungi in nasal mucus has been established by different groups, but so far it has not been shown how the immune system could even recognize such extramucosal—extracorporal—fungal targets. The aim of this study was to determine whether fungal DNA is present in tissue specimens taken from patients with polypoid CRS. Methods Twenty-seven surgical specimens were collected from patients suffering from CRS. Fifteen surgical specimens from healthy ethmoidal mucosa served as controls. A second set …

0301 basic medicineAdultMalePathologymedicine.medical_specialtyAdolescentChronic rhinosinusitis030106 microbiologyNose NeoplasmsMucous membrane of noseNose neoplasmPolymerase Chain Reactionlaw.invention03 medical and health sciencesNeuroma0302 clinical medicineNasal Polypslawotorhinolaryngologic diseasesMedicineHumansNasal polypsSinusitis030223 otorhinolaryngologySinusitisChildDNA FungalPolymerase chain reactionRhinitisbusiness.industryCase-control studyFungiFungal DNAMiddle Agedmedicine.diseaseNasal MucosaOtorhinolaryngologyCase-Control StudiesChronic DiseaseFemalebusinessAmerican journal of rhinology
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Transcriptome Analysis Identifies Doublesex and Mab-3 Related Transcription Factor (DMRT3) in Nasal Polyp Epithelial Cells of Patients Suffering from…

2021

Background: Aspirin-exacerbated respiratory disease (AERD) is a syndrome characterised by chronic rhinosinusitis, nasal polyps, asthma and aspirin intolerance. An imbalance of eicosanoid metabolism with anover-production of cysteinyl leukotrienes (CysLTs) has been associated with AERD. However, the precise mechanisms underlying AERD are unknown. Objective: To establish the transcriptome of the nasal polyp airway epithelial cells derived from AERD patients to discover gene expression patterns in this disease. Methods: Nasal airway epithelial cells were isolated from 12 AERD polyps and 8 AERD non-polyp nasal mucosa samples as controls from the same subjects. Utilising the Illumina HiSeq 2500 …

0301 basic medicineMaleMucous membrane of noseBiochemistryDMRT3TranscriptomeTranscription Factors TFII0302 clinical medicinetranscriptome analysisGene expressionMedicineNasal polypsRNA-SeqEicosanoid metabolismAnti-Inflammatory Agents Non-SteroidalMiddle AgedImmunohistochemistryQR1-502030220 oncology & carcinogenesisImmunohistochemistryFemalemedicine.symptomAdultLeukotrienesAspirin-exacerbated respiratory diseaseInflammationMicrobiologyArticle03 medical and health sciencesImmune systemNasal Polypsotorhinolaryngologic diseasesHumansSinusitisMolecular BiologySkin TestsAspirinbusiness.industryGene Expression Profilingnasal airwayEpithelial Cellsmedicine.diseaseRespiration Disorders030104 developmental biologyImmunologyChronic DiseaseNasal LavageAsthma Aspirin-InducedbusinessTranscriptomeBiomolecules
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Expression, Localization, and Significance of Vascular Permeability/Vascular Endothelial Growth Factor in Nasal Polyps

2005

Background The exact etiologic mechanisms leading to the formation of nasal polyps have remained largely obscure. A key phenomenon of this specific type of chronic inflammatory disease in nasal respiratory mucosa is remarkable edema. Vascular permeability/vascular endothelial growth factor (VPF/VEGF) plays an important role in inducing angiogenesis and modulating capillary permeability. Objective To study the expression and localization of VPF/VEGF as a putative key factor in nasal polyp development. Methods Specimens of nasal polyps (n = 12) were harvested during endonasal sinus surgery in patients with polypous chronic rhinosinusitis. Specimens of healthy nasal respiratory mucosa (n = 12)…

0301 basic medicinePathologymedicine.medical_specialty030102 biochemistry & molecular biologybusiness.industryVascular permeabilityChronic inflammatory diseasemedicine.diseaseVascular endothelial growth factor03 medical and health scienceschemistry.chemical_compound0302 clinical medicineOtorhinolaryngologychemistryotorhinolaryngologic diseasesMedicineNasal polyps030223 otorhinolaryngologybusinessAmerican Journal of Rhinology
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MUC4 impairs the anti-inflammatory effects of corticosteroids in patients with chronic rhinosinusitis with nasal polyps

2017

Background Current evidence suggests that membrane-tethered mucins could mediate corticosteroid efficacy, interacting with glucocorticoid receptor (GR) in patients with chronic rhinosinusitis with nasal polyps (CRSwNP). Mucin 4 (MUC4)–tethered mucin is expressed in nasal polyp (NP) epithelial cells and upregulated under inflammatory conditions. Moreover, MUC4β has the capacity to interact with other intracellular proteins. We hypothesized that MUC4 modulates corticosteroid efficacy of patients with CRSwNP. Objective We sought to analyze the role of MUC4 in corticosteroid effectiveness in different cohorts of patients with CRSwNP and elucidate the possible mechanisms involved. Methods Eighty…

AdultMale0301 basic medicineSmall interfering RNAmedicine.drug_classImmunologyAnti-Inflammatory AgentsDrug ResistanceBiologyDexamethasoneCell LineYoung Adult03 medical and health sciencesNasal PolypsGlucocorticoid receptorPregnenedionesmedicineglucocorticoid receptorHumansImmunology and AllergyNasal polypsSinusitisCells CulturedDexamethasoneAgedRhinitisnasal polypGene knockdownMucin-4chronic rhinosinusitisMucincorticosteroid resistanceEpithelial CellsMiddle Agedmedicine.diseaseHEK293 Cells030104 developmental biologyMUC4Chronic DiseaseImmunologyCorticosteroidImmunohistochemistryFemalesense organsmedicine.drug
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Notch‐1 signaling activation sustains overexpression of interleukin 33 in the epithelium of nasal polyps

2019

Abstract BACKGROUND: Alterations in the nasal epithelial barrier homeostasis and increased interleukin 33 (IL-33) expression contribute to the pathogenesis of chronic rhinosinusitis with nasal polyps (CRSwNP). AIMS: As Notch-1 signaling is crucial in repair processes of mucosa, the current study assessed Notch-1/Jagged-1 signaling and IL-33 in the epithelium of nasal polyps biopsies from allergic (A-CRSwNP; n = 9) and not allergic (NA-CRSwNP; n = 9) subjects by immunohistochemistry. We also assessed, in a model of nasal epithelial cells, the effects of stimulation of Notch-1 with Jagged-1 on the expression of IL-33 (by flow cytometry, immunofluorescence, and immunocytochemistry), Jagged-1 (…

AdultMale0301 basic medicineendocrine systemPhysiologyClinical BiochemistryImmunocytochemistryStimulationBiologyCell LineFlow cytometryYoung Adult03 medical and health sciencesNasal Polyps0302 clinical medicinestomatognathic systemmedicineHumansNasal polypsPhosphorylationReceptor Notch1SinusitisCyclic AMP Response Element-Binding ProteinNotch 1medicine.diagnostic_testEpithelial CellsCell BiologyMiddle AgedInterleukin-33medicine.diseaseRhinitis AllergicMolecular biologyEpitheliumUp-RegulationInterleukin 33Nasal Mucosa030104 developmental biologymedicine.anatomical_structure030220 oncology & carcinogenesisChronic DiseaseIL-33; Notch-1; chronic rhinosinusitis; nasal epithelium; nasal polypsImmunohistochemistryFemaleNOTCH-1 INTERLEUKIN 33 NASAL POLYPSJagged-1 ProteinSignal TransductionJournal of Cellular Physiology
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No evidence for a correlation of glutathione S-tranferase polymorphisms and chronic rhinosinusitis.

2011

OBJECTIVE: Cellular detoxification mechanisms are mandatory for cellular protection against oxidative stress and reactive oxygen species. One major group of antioxidative active enzymes involved in cellular detoxification are the Glutathione S-Transferases (GST). Multiple subtypes like GSTM1, GSTP1, and GSTT1 and variants of them are known, arising from allelic variations of the GST loci. Moreover, functional variants occur in high percentages and have been associated with diseases like bronchial asthma and bronchial hyperresponsiveness. The interplay of oxidative stress, detoxifying genes like GSTs and the genesis of respiratory tract illness is under contradictory debate. In this study, w…

AdultMaleAllergyCellular detoxificationComorbidityGSTP1Nasal Polypsotorhinolaryngologic diseasesmedicineGenetic predispositionHypersensitivityHumansNasal polypsGenetic Predisposition to DiseaseSinusitisAsthmaGlutathione TransferaseRhinitisPolymorphism Geneticbusiness.industryGeneral Medicinerespiratory systemmedicine.diseaseAsthmaNasal MucosaOxidative Stressmedicine.anatomical_structureOtorhinolaryngologyGlutathione S-Transferase piBronchial hyperresponsivenessImmunologyChronic DiseaseFemalebusinessRespiratory tractRhinology
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Aspirin Intolerance in Patients with Chronic Sinusitis

1999

Aspirin intolerance in patients with chronic sinusitis is often a cause of early recurrence of symptoms after surgical treatment. This study assesses 84 patients who were tested for acetylsalicylic acid intolerance after presenting with symptoms like chronic rhinosinusitis, sometimes bronchial asthma, coexisting allergies or a history of aspirin sensitivity. Nasal polyposis was found in a majority of cases, often recurrent after previous surgery. The levels of eicosanoids such as peptido-leukotrienes and prostaglandin E<sub>2</sub> were analyzed in isolated blood cells and compared with a healthy control group. Aspirin-intolerant patients showed elevated basal levels of peptido-…

AdultMaleLeukotrienesmedicine.medical_specialtyAllergymedicine.medical_treatmentGastroenterologyAntibodiesDinoprostoneDrug HypersensitivityNasal PolypsInternal medicinemedicineHumansAntipyreticSinusitisSinusitisAsthmaChemotherapyAspirinAspirinbusiness.industryAnti-Inflammatory Agents Non-SteroidalChronic sinusitisMiddle Agedmedicine.diseaseOtorhinolaryngologyEicosanoidAnesthesiaChronic DiseaseFemalebusinessmedicine.drugORL
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Increased in vitro cvsteinvl leukotriene release from blood leukocytes in patients with asthma, nasal polyps, and aspirin intolerance

1996

In vitro cysteinyl leukotriene (cLT) release from blood leukocytes was measured in eight normal individuals (NI), nine patients with nasal polyps (NP) without aspirin intolerance, and eight patients with NP, asthma, and aspirin intolerance (AI). Blood leukocytes were prestimulated with interleukin-3 (IL-3) and incubated with acetylsalicylic acid (ASA) (10 and 100 micrograms/ml) together with C5a (10(-8) mol/l) for 18 h. cLT release (LTC4, LTD4 and LTE4) from blood leukocytes was measured with a competitive enzyme-linked immunoassay. Background cLT release was 259 +/- 66 pg/ml (mean +/- SEM) in the NI group, 185 +/- 33 pg/ml in the NP group, and 578 +/- 136 pg/ml in the AI group (P = 0.1). A…

AdultMaleLeukotrienesmedicine.medical_specialtyLeukotriene B4ImmunologyDrug Hypersensitivitychemistry.chemical_compoundNasal PolypsInternal medicineLeukocytesmedicineHumansImmunology and AllergyNasal polypsIncubationAsthmaLeukotriene E4AspirinLeukotriene C4business.industryMiddle Agedmedicine.diseaseAsthmaIn vitroEndocrinologychemistryToxicityImmunologyFemalebusinessAllergy
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Proliferative Activity and Cytometric Characteristics in Polyps of the Nasal Cavity and Paranasal Sinuses

2000

Although several investigations have revealed the influence of cytokines, allergy, and environmental factors in polyp development, the etiology of nasal polyps is still unknown. To estimate the biology of this common disease the operative specimens of 50 patients who underwent surgery for polyps of the nasal cavity and the paranasal sinuses were examined; of these, 10 patients had recurrent disease and 23 patients had an allergy. The investigations included routine histology and quantitative DNA measurements, along with immunohistochemical identification of proliferation markers (i.e., MIB-1; proliferating cell nuclear antigen, PCNA). Histologically, most polyps revealed an infiltration wi…

AdultMaleNasal cavityParanasal Sinus NeoplasmPathologymedicine.medical_specialtyAdolescentAneuploidy03 medical and health sciencesNasal PolypsPolyps0302 clinical medicineProliferating Cell Nuclear Antigenotorhinolaryngologic diseasesmedicineHumansNasal polypsChild030223 otorhinolaryngologyAgedPloidiesbiologybusiness.industryHistologyDNA NeoplasmMiddle Agedmedicine.diseasedigestive system diseasesSquamous metaplasiaProliferating cell nuclear antigenmedicine.anatomical_structureParanasal sinusesOtorhinolaryngology030220 oncology & carcinogenesisbiology.proteinFemalebusinessCell DivisionParanasal Sinus NeoplasmsAmerican Journal of Rhinology
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A Passive-Marker-Based Optical System for Computer-Aided Surgery in Otorhinolaryngology: Development and First Clinical Experiences

1999

Objectives: To develop a new type of optical computer-aided surgery (CAS) device that overcomes some of the restrictions of common systems and to examine its accuracy and usability under laboratory and intraoperative conditions. Study Design: Prospective study using laboratory experiments and intraoperative data collection. Methods: An optical CAS system applying passive optical markers for coordinate determination was developed. Laboratory accuracy measurements were obtained on a Plexiglas model with known coordinates of fiducial markers, before and after predefined table movements. Intraoperative accuracy measurements were recorded from 24 patients undergoing endonasal surgery of the para…

AdultMaleOptics and Photonicsmedicine.medical_specialtyEndoscopemedicine.medical_treatmentNasal PolypsParanasal SinusesmedicineHumansSinusitisbookMouthpieceComputer-assisted surgerybusiness.industryPassive markerComputer aided surgeryOtorhinolaryngologic Surgical ProceduresSurgeryImage-guided surgeryOtorhinolaryngologyOtorhinolaryngologyTherapy Computer-Assistedbook.journalFemaleFiducial markerNuclear medicinebusinessThe Laryngoscope
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